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Linformazione è la chiave di volta per le malattie rare
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Lettera aperta degli Omeopati FIAMO al Consiglio Regionale, alla Giunta Regionale e ai Partiti politici della Regione Lazio
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Ferruccio Fazio ha visitato il Centro Clinico NEMO
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Mario Melazzini sul testamento biologico
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Testamento biologico. Iardino: "Sia rispettata la volontà dei pazienti"
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Anche le cellule passano le frontiere - Tavola Rotonda, mercoledì 11 marzo 2009, ore 10,30
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Autismo, iperattività, epilessia, metabolismo, tossicologia, nutrizione e riabilitazione: approccio biologico e terapeutico
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Può la Programmazione Neuro Linguistica migliorare la complìance?
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X° Congresso Nazionale SICOP
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Uroandrologia Ricostruttiva
Cellula cancerosa
AMAVAS
Onlus For a smile
Semeiotica biofisica
Chirurgia Laser
Otorinolaringoiatria
Prevenzione prostata
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Home Rassegna stampa Ultime pubblicate Associazione tra il consumo di cibi ricchi di flavonoli, flavoni e flavonoidi e il rischio di cancro in un campione di donne anziane e di mezza età |
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Associazione tra il consumo di cibi ricchi di flavonoli, flavoni e flavonoidi e il rischio di cancro in un campione di donne anziane e di mezza età |
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Background: Flavonoids may protect against cancer development through several biological mechanisms. However, epidemiologic studies on dietary flavonoids and cancer risk have yielded inconsistent results.
Objective: We prospectively investigated the association between the intake of selected flavonoids and flavonoid-rich foods and risk of cancers in the Women's Health Study.
Design: A total of 3234 incident cancer cases were identified during 11.5 y of follow-up among 38,408 women aged 45 y. Intake of individual flavonols (quercetin, kaempferol, and myricetin) and flavones (apigenin and luteolin) was assessed from food-frequency questionnaires. Cox regression models were used to estimate the relative risk (RR) of total and site-specific cancer across increasing intakes of total and individual selected flavonoids and flavonoid-rich foods (tea, apple, broccoli, onion, and tofu).
Results: The multivariate RRs of total cancer across increasing quintiles of total quantified flavonoid intake were 1.00, 1.00, 0.93, 0.94, and 0.97 (P for trend = 0.72). For site-specific cancers, the multivariate RRs in the highest quintile of total quantified flavonoid intake compared with the lowest quintile were 1.03 for breast cancer, 1.01 for colorectal cancer, 1.03 for lung cancer, 1.15 for endometrial cancer, and 1.09 for ovarian cancer (all P > 0.05). The associations for the individual flavonoid intakes were similar to those for the total intake. There was also no significant association between intake of flavonoid-rich foods and the incidence of total and site-specific cancers.
Conclusion: Our results do not support a major role of 5 common flavonols and flavones or selected flavonoid-rich foods in cancer prevention.
American Journal of Clinical Nutrition 2009, vol. 89, n. 3, pp. 905-912
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